Textbook of Special Pathological Anatomy of Domestic Animals by Paul Cohrs

By Paul Cohrs

Textbook of distinctive Pathological Anatomy of household Animals shouldn't be looked in basic terms as a textbook for college kids, yet really as one that can also be of suggestions to them of their later paintings, in addition to to veterinarians more often than not operating within the quite a few branches of the occupation, within the evaluation of pathological alterations. As many new ailments became identified and masses new info in terms of pathogenesis and aetiology has been won because the visual appeal of the final variation, radical revision of the booklet has develop into primary. The discussions during this publication disguise the circulatory method, blood-forming organs, reticulo-endothelial method, breathing procedure, digestive organs, peritoneum, apprehensive method, urinary organs, genital organs, organs of locomotion, endocrine glands, and epidermis.

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Textbook of Special Pathological Anatomy of Domestic Animals

Textbook of certain Pathological Anatomy of household Animals shouldn't be looked basically as a textbook for college kids, yet quite as one that can also be of counsel to them of their later paintings, in addition to to veterinarians mostly operating within the a variety of branches of the career, within the evaluate of pathological alterations.

Extra resources for Textbook of Special Pathological Anatomy of Domestic Animals

Sample text

In aortic stenosis the blood is dammed back by the narrowed orifice. The left ventricle hypertrophies but the effects of back pressure may be noticeable even further back in the left atrium and in the lungs. In aortic incompetence blood FIG. 41. Stenosis of left atrioventricular orifice as a result of bacterial endocarditis. Seen from the atrium. flows back into the left ventricle during diastole. The latter becomes dilated and undergoes compensatory hypertrophy. Pulmonary stenosis leads to hypertrophy of the right ventricle with build-up of pressure further back in the right atrium and in the greater circulation.

These foci are seen to lie in slightly denser fibrosed areas. In pronounced cases most of the wall of the right atrium is transformed into a mass of stout, sometimes compact sometimes spongy, bone tissue, which is as hard as true bone. The remarkable thing is that most of the cases hitherto observed have exhibited no clinical signs of cardiac involvement. Microscopically, the basis of the whole condition is seen to be a chronic granulation tissue, poor both in cells and vessels, which has caused much atrophy of the muscle fibres, and in which miliary tuberculosis or rheumatoid cell granulomas, and sometimes also a peculiar granulating endangitis obliterans can be observed.

Macroscopically, the myocardium, like the skeletal muscles, exhibits multiple, dark-red, soft and friable foci, often containing gas bubbles. Microscopically, there is granular disintegration and hyaline degeneration of muscle fibres, interstitial haemorrhages and signs of inflammatory oedema of greater or less intensity. A myocarditis with multiple focal lesions can also develop in swine fever, the foci exhibiting the hyaline necrosis of vessel walls, first observed by Röhrer. ALLERGIC MYOCARDITIS A serous or serous-proliferative myocarditis, which may terminate in scar formation, is seen particularly in streptococcal infections of horses, but it also occurs in other domestic animals.

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