Chemical Contraception by John P. Bennett (auth.)

By John P. Bennett (auth.)

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The fruits of this biological insight were not realized in contraception for some time, the intervening World War II doing much to slow progress in research. Pincus (1955), among others, later showed that progesterone in 300-mg doses given daily from day 5 through day 25 of the menstrual cycle inhibited ovulation. , bleeding occurring before the time of expected menses) was a problem. Even when estrogen was given together with progesterone to prevent 24 COMBINATION AND SEQUENTIAL ORAL CONTRACEPTIVES the breakthrough bleeding, the combination sooner or later failed to control menstruation and ovulation (Whitelaw, 1955).

All animals were killed on day 15 of pregnancy. b All dose groups contained three animals, which were Inseminated with > 1 07 spermatozoa. Source: Vickery and Bennett (1969, table 4). penetration of the cervical mucus was observed up to 2 hours after dosing, but there was virtually no linear progression of spermatozoa after 4 hours. Penetration of spermatozoa through the cervical mucus occurred again at 22-24 hours after dosing. 5 mg megestrol acetate. Complete fertility control was obtained. Some of the social and functional problems of using a precoital contraceptive may explain the lack of development of this method.

0-mg dose. l:re observed in some women. Later studies by Connell (1966), MartinezManautou et al. 5-mg chlormadinone acetate minipill. They also showed that the scant, viscous cervical mucus in the progestin-treated women was hostile to penetration by ejaculated spermatozoa. The production of an impenetrable, hostile cervical mucus by administration of low doses of progestins was confirmed for several different steroids by Mason et al. (1967), Roland (1968). Zanartu et al. 4 Summary of dosing schedules, time elapsing before animals were killed, and observations made at time of death.

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